Rheumatism ntawm txhais ceg yog hais txog cov kab mob ntawm oob khab autoimmune. Tus kab mob no yog los ntawm ib qho kev mob nkeeg ntawm cov leeg, leeg nqaij thiab mob plawv nres los ntawm tus kab mob no feem ntau los ntawm tus mob streptococci. Feem ntau cov pob qij txha (lub hauv caug, pob ntseg, pob luj taws) muaj feem xyuam nrog cov kab mob pathological, tab sis cov pob qij txha, piv txwv, cov ntiv taw muaj qee zaum cuam tshuam.
Ua rau rheumatism
Txoj kev loj hlob ntawm kev mob rheumatic nyob rau hauv lub cev yog provoked:
- nkag mus ntawm cov kab mob pathogenic mus rau cov pob qij txha nrog ntshav khiav;
- ib qho kev tsis haum tshuaj rau co toxins tso tawm los ntawm cov kab mob.
Rheumatism yog ib qho teeb meem tom qab muaj pes tsawg tus kab mob kis, xws li:
- angina;
- liab plab liab ;
- o ntawm txoj hlab pas nqhis;
- duav, thiab lwm yam.
Cov cim qhia ntawm ceg rheumatism
Cov cwj pwm ntawm cov kab mob rheumatism hauv cov laus yog:
- mob tshwm sim ntawm kev sib txawv thiab ntev;
- liab liab thiab o nyob rau hauv lesion, lub npe hu ua cov nyhuv ntawm "daim tawv nqaij ci";
- nce lub cev kub, tshwj xeeb tshaj yog cov lus hais nyob rau thaj tsam ntawm cov pob txha.
- mob nyob hauv txoj haujlwm ntawm lub plawv mob;
- tag nrho deterioration ntawm tus mob (poob qab los noj mov, poob ntawm lub zog, pw ntxaug, lwm yam).
Yog xav paub! Rau rheumatism ntawm ob txhais ceg yog tsiag ntawv los ntawm ib tug symmetrical lesion ntawm pob yas qej txha.
Thaum kuaj xyuas tus neeg mob, tus kws tshawb fawb qhia:
- nce ntim hauv pob ntseg hauv tsev;
- muaj cov cim qhia ntawm tus mob;
- cov kev hloov tsis zoo hauv cov leeg cov leeg.
Yog hais tias kev kho tsis tau pib rau lub sij hawm, tus neeg mob yuav poob qoj cev, thiab hloov tsis tau nyob hauv cov pob txha hauv pob txha: Cov pob zeb ntxhia raug ntxuav tawm thiab cov pob txha ua kom ntxub.
Kev kho ntawm ko taw ob leeg rheumatism
Tus neeg mob uas muaj rheumatism, raws li txoj cai, muaj kev kho mob hauv tsev kho mob. Thaum thawj theem ntawm kev kho, pw so tau qhia nrog kev kho mob siab, thaum tus neeg mob tus mob zoo dua, qhov kev tawm dag zog yog lus.
Kev kho mob ntawm kev qoob loo ntawm cov pob qij txha ntawm cov taw thiab lwm qhov ntawm cov ceg yog cov kev tshem tawm cov tsos mob thiab nrog rau kev siv cov tshuaj xws li:
1. Cov ntsiav tshuaj nrog cov nyhuv (Tshuaj Aspirin, Paracetamol).
2. Cov tshuaj tsis-steroidal anti-inflammatory drugs:
- parenteral administration (indomethacin, diclofenac);
- kev siv sab nraud (indomethacin, diclofenac, fastum gel);
- qhov ncauj tswj hwm (Ibuprofen, Piroxicam, Indomethacin, Naproxen, Diclofenac, Lornoxicam).
3. Cov kab mob glucocorticoid (Hydrocortisone, Prednisolone, Dexamethasone).
4. Antibiotics (Nruas, Amikacin).
5. Immunosuppressants (Cyclosporin A, Cyclophosphamide, Methotrexate).
6. Vitamin-pob zeb hauv av complexes.
7. Mob plab glycosides (Pumpan, Tonginal) thiab kev npaj muaj potassium, magnesium (Asparcum, Panangin).
Tom qab tshem cov cim ntawm mob leg rheumatism, cov txheej txheem physiotherapeicic raug muab kho rau kev kho mob:
- UHF;
- cov ntawv thov paraffin;
- electrophoresis thiab lwm yam.
Tsis tas li ntawd, tus kws kho mob yuav tsum tau xaiv lub plasmapheresis - ib txoj kev los ntxuav cov ntshav ntawm co toxins thiab tshuaj tua kab mob.
Nws yog ib qho tseem ceeb kom ua raws li kev noj haus zoo rau cov khoom muaj protein ntau, tab sis muaj cov roj tsawg thiab cov roj tsawg.
Nrog rau kev kho mob rau kev tshem tawm cov cim qhia txog kev ua npaws rheumatic nyob rau hauv kev kho mob ntawm pej xeem cov kev pab cuam. Piv txwv, siv propolis ncuav, propolis alcoholic infusion, ointment los ntawm ib tug tov ntawm propolis thiab petrolatum, tincture ntawm aconite. Txo o thiab txo qhov mob ntawm turmeric. Tsis zoo, iodine-ntsev compresses, kev siv ntawm cov av nplaum xim. Rheumatologists tau qhia tias noj cov txiv hmab txiv ntoo tshiab ntawm cranberries, blueberries, cranberries los yog haus dej kua txiv hmab txiv ntoo txhua hnub.